OUR VIEW
Mainstream medicine fails to address chronic and autoimmune diseases due to wrong medical paradigm and wrong delivery model
Chronic Inflamation Is a sign and not a cause of Disease
Inflammation refers to your body’s process of fighting against things that harm it, such as infections, injuries, and toxins, in an attempt to HEAL ITSELF.
Mainstream Medicine Today
When it comes to chronic inflammation you will be told that your immune system mistakenly attacks your own cells and tissues. This process is called molecular mimicry. The big problem with this is the fact that molecular mimicry is only a theoretical possibility that sequence similarities between foreign and self-peptides are sufficient to result in autoimmunity. Does this really mean all conventional treatments are based on theoretical possibility?
Yes, unfortunately it does and the result is endless flavor of prescribed drugs targeting the inflammation itself, resulting in little to moderate and temporary symptoms relief with statistically bad long-term prognosis.
Molecular mimicry or intra-cellular “stealth” infections
In the era of Human Microbiome project one thing bacame clear – the human body is a superorganism in which thousands of microbial genomes continually interact with the human genome. A range of physical and neurological inflammatory diseases are now associated with shifts in microbiome composition. Seemingly disparate inflammatory conditions arise from similar disruption of microbiome homeostasis. A really good such example is crohn’s disease currently being treated with immunosuppression while recent studies confirm FTM (fecal microbiota transplant) theraphy is definately the better choice of treatment.
It is now understood that the vast majority of microbes capable of persisting in Homo sapiens cannot be cultured in a laboratory. In contrast, studies using these new molecular tools have revealed the presence of thousands of previously unknown microbes in human tissue and blood. These microbes persist both in and on the human body, and are collectively referred to as the human microbiome. Many of these newly characterized microbes have the capacity to contribute to human disease processes. For example, the amniotic fluid is widely regarded as being sterile. However, in 2010, DiGiulio and Relman and their colleagues at Stanford University published a seminal study that used molecular methods to identify eighteen different bacterial taxa in the amniotic fluid of woman.
Where you find inflammation there is successive infection with few exceptions.
It is becoming increasingly clear that inflammatory disease processes are not due to acquisition of any single pathogen. Instead, they appear to result from alterations in the complex microbial communities. It follows that Koch’s postulates, which dictate that one microbe must be proven causative of a single disease state, can no longer be supported in the era of the metagenome.
Many of the pathogens that contribute to chronic inflammatory disease persist inside the nucleated cells. These intracellular pathogens directly interfere with transcription, translation, and DNA repair at the cellular level. As the concept of “autoimmunity” is re-evaluated in light of chronic infection, the utility of immunosuppression must be reconsidered. Immunosuppressive therapies for inflammatory disease may provide short-term relief by slowing the cytokine and chemokine release associated with a healthy immune response towards acquired pathogens. However, pathogens are able to spread with much greater ease over the long term, leading to relapse and instability. Indeed, during the period that immunosuppressive therapies have become the standard of care, the incidence of nearly every chronic disease increased dramatically.
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